Processed foods probably aren't good for you.

Canned soup has way too much salt.

I always wonder how much I'm getting each time I handle a receipt.

This is bad news for my zombie apocalypse food pantry.

But this means your shelf life will be tripled.

PsyLord: This is bad news for my zombie apocalypse food pantry.

The whole BPA scare is way overblown. Dose makes the poison.

DO NOT ADD WATER!

Was it Hormel?

Egh, I have been making my own soups from scratch for years now.

Soup is really, REALLY easy to make. Water. Veggies. Meat of choice. Spices. Throw in some noodles near the end.

Canned soup is nasty :/

this is retarded and sketchy for a few reasons: it was lame for them to name a soup maker when it isn't the soup but the lining in the cans. i wonder who it was at Progresso that pissed off the researchers doing the study. and if they're focusing on the cans and not the product inside, it could have been a five-day study of people who ate chili, fruit, beans or anything that comes out of a can. also, if BPA is found in dental sealants, then this study was a waste of time and money. oh well, huh.

we're gonna die anyway, so what's the point. until i find a good recipe for Albondigas, i'm eating it out of a can.

Useless article is useless. What are the actual levels? What levels are dangerous?

If I have a millionth of a penny and someone gives me an entire penny, it doesn't make me rich even though my total wealth has increased by a huge percentage.

So am I to understand that if I have a picogram of bpa in my blood, this will raise my blood level to one nanogram?
THE FARKING HORROR!!1!1!

I think the question that everyone should be asking is JUST WHY THE FRAK IS BPA IN THE CAN TO BEGIN WITH? It's not "good" for living things in ANY dose, just less harmful in smaller ones.

Raspil: this is retarded and sketchy for a few reasons: it was lame for them to name a soup maker when it isn't the soup but the lining in the cans. i wonder who it was at Progresso that pissed off the researchers doing the study. and if they're focusing on the cans and not the product inside, it could have been a five-day study of people who ate chili, fruit, beans or anything that comes out of a can. also, if BPA is found in dental sealants, then this study was a waste of time and money. oh well, huh.

we're gonna die anyway, so what's the point. until i find a good recipe for Albondigas, i'm eating it out of a can.

Wrong. Shame the companies. The exposure will cause other manufacturers to take a look at the practice.

This anonymous secret shiat needs to end. Transparency and responsibility are today's secret words.

Do we naturally have a BPA level? Cuz last I checked, 0 x 1000...

A lot of these studies boil down to "we have some amazing new techniques that move our sample range out of "none that we can detect" to "we can now detect incredibly small amounts, well below the toxicity level, so we'll put out some bad science to fill out a thesis in Public Health."

All y'all's a chromosome short and a year late: here comes the witchcraft. (new window)

parkthebus: 1000% increase from what? Anyone know the analytical method/detection limit of BPA?

Don't believe the hype.

jtown: Useless article is useless. What are the actual levels? What levels are dangerous?

If I have a millionth of a penny and someone gives me an entire penny, it doesn't make me rich even though my total wealth has increased by a huge percentage.

Tainted1: So am I to understand that if I have a picogram of bpa in my blood, this will raise my blood level to one nanogram?
THE FARKING HORROR!!1!1!

It's almost like there is a PR damage control team dedicated to Fark.

Also...1000% in contrast the the levels at which a typical person is exposed to in the course of their daily, non-canned soup consuming, life. It isn't about the contents though; it is about the packaging materials and process.

It just so happens that heating a slightly corrosive substance to a high temperature and sealing it in an air tight container can result in some unexpected levels of erosion. Be it canned soup, diet soda or pork brains in gravy, the result is still the same: abusive consumption will give you man-tits (moobies).

All of this is irrelevant because I said biatch make me a sammich!

odinsposse: PsyLord: This is bad news for my zombie apocalypse food pantry.

[profile.ak.fbcdn.net image 200x200]

A humorous image. Thank you.

/Caucasian-American

Tainted1: So am I to understand that if I have a picogram of bpa in my blood, this will raise my blood level to one nanogram?
THE FARKING HORROR!!1!1!

No, it would raise it to 11 picograms, or .011 nanograms.

1000% more, not 1000 times.

shijjiri:
It's almost like there is a PR damage control team dedicated to Fark.

Yes, the actual professional scientists have hired us to counter the horrible negative PR generated by people who do studies like this one, to counter the anti-science people like CSPI and the guy who wrote this silly thing up for his doctoral thesis in public health.

One of the big projects they've got us working on is teaching people that any study that harps on relative numbers ("100% urinary BPA") while not mentioning that the actual numbers are mind-bogglingly low is almost certainly not based on pretty bad science.

My SIL, who doea a lot of home cannings, was worried about the dangers of BPA, based on what she read in the magazines at the checkout line in the holistic, vegan, communist, food cooperative where she buys her free-range mung beans in bulk so, I did a little research and sent her the following letter:


Hi (NAME REDACTED):

I haven't forgotten your questions about BPA. I didn't want to answer off-the-cuff without doing a little digging, which is what took me so long....
Anyway, here's what I came up with...

Bisphenol A is one of the starting materials used in the synthesis of the varnish that is coated inside of cans to prevent interaction with the metal. Not all of the starting material is converted to final product, so the varnish coating contains small amounts of of precursors which can leach out of the varnish and into the canned food. I've done a literature search and have compiled a list of the abstratcts from several of the most recent papers. Almost all of these are concerned with migration of BPA from cans and do not address canning in glass with coated lids.

There is really no hard data that quantifies the health risk to humans from BPA. We know that if you feed it to rats, it can cause a number of serious problems. Paper 1 states that when rats were given 5 milligrams per kilogram of body weight per day, no adverse effects were observed even after several generations. So, it's possible to set a "safe"consumption limit for rats.

What about people? Well, you could do the same thing to say, college students, or lawyers, but their mothers wouldn't like it. So, it would appear as if the regulatory agencies in the US and Europe have simply take the rat numbers and divided by 100, setting the "tolerable daily intake" of BPA at 0.05 mg/kg of body weight per day.


Math time:

A 110-lb person weighs 50 kilograms, so the maximum "safe" amount they can consume would be:

(0.05 milligrams/kg body weight/day) *(50 kilograms)= 2.5 mg/day.


2.5 mg is not a lot, so the next question is,"how much BPA are you exposed to?" Paper 3 gives an estimate of median exposure in the US by measuring BPA in peoples' urine; they come up with a value of 34 ng/kg per day; one nanogram [ng] is a millionth of a mg. Referring back to our 50 kg person, that works out to about 1/1500th of the "safe" level of 2.5 mg. A similar study in Belgium came up with roughly the same calculated exposure levels (Paper 4)

A more direct way to determine exposure is to measure BPA concentrations in canned food, as in papers 5 and 6 below. If you know the levels in various foods, you can estimate how much you eat, and, in a perfect world, figure out your exposure. Unfortiunatelly, measured concentrations vary widely among different canned foods; some studies have reported differing levels in different cans of the same product/lot.

However, a couple of trends seem to hold true across studies:

More BPA is leached into foods at higher temperatures.

Canned fish seemed to have the highest levels, although one paper [2] states they were low

Little or no BPA was seen in food stored in glass; very low levels were seen in beverages stored in PET bottles,

Papers 5 and 6 list the measured concentration of BPA in a variety of canned goods. Using these values, I have put together the following list which states how much of each food product you would have to eat every day to hit the maximum daily limit of 0.05mg/kg-day

Canned fish 24 kilograms (52 lbs)


Canned corn 30 kilograms (66 lbs

Canned soup 57-114 kilograms (125-250 lbs)

Baked beans 106 kilograms (234 lbs)

Peas 149 kilograms (327 lbs)

Evaporated milk 163 kilograms (350 lbs)

Luncheon meat 238 kilograms (524 lbs)

Infant formula 909 kilograms (2000lbs)

Tomato paste 1190 kilograms(2619 lbs)

So, to get back to the original question of "Should I worry about BPA leaching from canning lids?" I would say, no.


First of all, the values listed above are for foods stored in varnished cans, where there is more surface area to leach BPA from. Since your foodstuffs are only in minimal contact with the canning lids, you would have much less exposure.


Second, your family and, I assume, everyone likely to eat your delicious canned products do not eat much, if any canned food, which is where the bulk of BPA exposure comes from.

Obviously, it is always good to minimize exposure to potentially dangerous compounds,, and considering your lifestyle, you really have little to fear. Part of the hysteria about these "periodic scare-du-jours" is the fact that analytical instrumentation has become incredibly sensitive, selective and robust, meaning that more and more people are doing analyses of lots of things that they couldn't do before. Any swinging dick with a mass spectrometers can quantitate just about any molecule to concentrations in the nanogram (1 x 10-9g) to picogram (1 x 10-12g) level. If you know what to look for, the odds are that you will find it. This goes for other things such as plasticizers like Phthalic acid, dioctyl phthalate, and other stuff. They're everywhere, and have been for years.


So it's not so much that we've recently been assaulted by an evil plot to poison us, so much as we can now routinely and easily detect things at levels we couldn't before. I suspect there are people making political and/or economic hay off of environmental scares, as well as a large number of people who are not very discerning consumers of information.


Anyway, that's my sermon on BPA. If any of the following abstracts interest you, let me know, and I can pull up a copy of the complete articles.

Bibliography:

1. Scientific opinion on Bisphenol A: evaluation of a study investigating its neurodevelopmental toxicity, review of recent scientific literature on its toxicity and advice on the Danish risk assessment of Bisphenol A

By: Anadon, Arturo; Binderup, Mona-Lise; Bursch, Wilfried; Castle, Laurence; Crebelli, Riccardo; Engel, Karl-Heinz; Franz, Roland; Gontard, Nathalie; Haertle, Thomas; Husoy, Trine; Jany, Klaus-Dieter; Leclercq, Catherine; Lhuguenot, Jean-Claude; Mennes, Wim; Milana, Maria Rosaria; Pfaff, Karla; Svensson, Kettil; Toldra, Fidel; Waring, Rosemary; Wolfle, Detlef; Bell, David; Granum, Berit; Hass, Ulla; Holene, Edel; Mantovani, Alberto; Nygaard, Unni Cecilie; Parker, Terry; Reuter, Ulrike; Testai, Emanuela; Castoldi, Anna F.; Bashir, Saghir; Ciccolallo, Laura; Croera, Cristina; Oberg, Tomas; Theobald, Roberta Pinalli Anne; Verloo, Didier; EFSA Panel on food contact materials, enzymes, flavourings and processing aids CEF

Bisphenol A (BPA) is used in the manuf. of plastics, to produce reusable drinking bottles, infant feeding bottles and other food storage containers. EFSA was asked to evaluate a dietary developmental neurotoxicity study in rats and recent scientific literature (2007-2010) in terms of relevance for the risk assessment of BPA. The impact of these studies on the current Tolerable Daily Intake (TDI) of 0.05 mg BPA/kg body wt. (b.w.)/day as set by EFSA in 2006 was assessed. Advice on the Danish risk assessment underlying the Danish ban of BPA in food contact materials for infants aged 0-3 years is included. Overall, based on this comprehensive evaluation of recent toxicity data, the Panel on food contact materials, enzymes, flavourings and processing aids (CEF) concluded that no new study could be identified, which would call for a revision of the current TDI. This TDI is based on the No-Obsd.-Adverse-Effect-Level (NOAEL) of 5 mg/kg b.w./day from a multi-generation reproductive toxicity study in rats, and the application of an uncertainty factor of 100. This factor is regarded as conservative based on all information on BPA toxicokinetics. The Panel noted that some studies conducted on developing animals have suggested other BPA-related effects of possible toxicol. relevance, in particular biochem. changes in brain, immune-modulatory effects and enhanced susceptibility to breast tumors. These studies had several shortcomings. At present the relevance of these findings for human health cannot be assessed. Should any new relevant data become available in the future, the Panel will reconsider this opinion. A minority opinion is expressed by a Panel member and presented in an Annex to this opinion.


2. Concentration of Bisphenol A in Highly Consumed Canned Foods on the U.S. Market
By: Noonan, Gregory O.; Ackerman, Luke K.; Begley, Timothy H.

Metal food and drink cans are commonly coated with epoxy films made from phenolic polymers produced from bisphenol A (BPA). It is well established that residual BPA monomer migrates into can contents during processing and storage. While a no. of studies have reported BPA concns. in foods from foreign markets and specialty foods on the U. S. market, very few peer-reviewed data for the BPA concns. in canned food from the U.S. market were available. This study quantified BPA concns. in 78 canned and 2 frozen food products from the U.S. market using an adaptation of a previously reported liq. chromatog.-tandem mass spectrometry method. The tested products represented 16 different food types that are from the can food classifications that constitute approx. 65% of U.S. canned food sales and canned food consumption. BPA was detected in 71 of the 78 canned food samples but was not detected in either of the two frozen food samples. Detectable BPA concns. across all foods ranged from 2.6 to 730 ng/g. Large variations in BPA concns. were found between different products of the same food type and between different lots of the same product. Given the large concn. ranges, the only distinguishable trend was that fruits and tuna showed the lowest BPA concns. Expts. with fortified frozen vegetables and brine solns., as well as higher BPA concns. in canned food solids over liq. portions, clearly indicated that BPA partitions into the solid portion of foods.

3. Daily intake of bisphenol A and potential sources of exposure: 2005-2006 National Health and Nutrition Examination Survey
By: LaKind, Judy S.; Naiman, Daniel Q.

Nationally representative data on urinary levels of bisphenol A (BPA) and its metabolites in the United States from the 2005-2006 National Health and Nutrition Examn. Survey (NHANES) were used to est. daily BPA intakes. In addn., NHANES data on potential sources of BPA exposure and personal characteristics were explored for their assocn. with urinary BPA levels. On the basis of 2005-2006 NHANES urinary BPA data and assumptions described in this paper, median daily intake for the overall population is approx. 34 ng/kg-day. Median daily BPA intakes for men are statistically significantly higher than for women; there is a significant decrease in daily BPA intake with increasing age. Gender- and age-specific median intakes differ from the overall population by less than a factor of 2. Although ests. of daily BPA intake have decreased compared with those from the 2003-2004 NHANES, it is premature to draw conclusions regarding trends at this time, as there is no indication that BPA use declined from 2003 to 2006. On the basis of an assessment of urinary BPA and questionnaire data from the 2005-2006 NHANES, consumption of soda, school lunches, and meals prepd. outside the home - but not bottled water or canned tuna - was statistically significantly assocd. with higher urinary BPA. Journal of Exposure Science and Environmental Epidemiol. (2011) 21, 272-279; doi:10.1038/jes.2010.9; published online 17 March 2010.


4 Intake of bisphenol A from canned beverages and foods on the Belgian market
By: Geens, Tinne; Apelbaum, Tali Zipora; Goeyens, Leo; Neels, Hugo; Covaci, Adrian
Bisphenol A (BPA), a contaminant which may be present in the coating of cans, was detd. in 45 canned beverages and 21 canned food items from the Belgian market. Beverages had an av. BPA concn. of 1.0 ng/mL, while canned foods had a higher av. concn. of 40.3 ng/g. The amt. of BPA present in food items was dependent on the type of can and sterilization conditions rather than the type of food. For example, BPA was not detected in non-canned beverages (
5. Concentrations of bisphenol A in the composite food samples from the 2008 Canadian total diet study in Quebec City and dietary intake estimates
By: Cao, X.-L.; Perez-Locas, C.; Dufresne, G.; Clement, G.; Popovic, S.; Beraldin, F.; Dabeka, R. W.; Feeley, M.
A total of 154 food composite samples from the 2008 total diet study in Quebec City were analyzed for bisphenol A (BPA), and BPA was detected in less than half (36%, or 55 samples) of the samples tested. High concns. of BPA were found mostly in the composite samples contg. canned foods, with the highest BPA level being obsd. in canned fish (106 ng g-1), followed by canned corn (83.7 ng g-1), canned soups (22.2-44.4 ng g-1), canned baked beans (23.5 ng g-1), canned peas (16.8 ng g-1), canned evapd. milk (15.3 ng g-1), and canned luncheon meats (10.5 ng g-1). BPA levels in baby food composite samples were low, with 2.75 ng g-1 in canned liq. infant formula, and 0.84-2.46 ng g-1 in jarred baby foods. BPA was also detected in some foods that are not canned or in jars, such as yeast (8.52 ng g-1), baking powder (0.64 ng g-1), some cheeses (0.68-2.24 ng g-1), breads and some cereals (0.40-1.73 ng g-1), and fast foods (1.1-10.9 ng g-1). Dietary intakes of BPA were low for all age-sex groups, with 0.17-0.33 ug kg-1 body wt. day-1 for infants, 0.082-0.23 ug kg-1 body wt. day-1 for children aged from 1 to 19 years, and 0.052-0.081 ug kg-1 body wt. day-1 for adults, well below the established regulatory limits. BPA intakes from 19 of the 55 samples account for more than 95% of the total dietary intakes, and most of the 19 samples were either canned or in jars. Intakes of BPA from non-canned foods are low.

6. Bisphenol A in canned food products from Canadian markets


By: Cao, Xu-Liang; Corriveau, Jeannette; Popovic, Svetlana

A method based on solid phase extn. followed by derivatization and gas chromatog.-mass spectrometry anal. was validated for the detn. of bisphenol A (BPA) in canned food products. This method was used to analyze 78 canned food products for BPA. Concns. of BPA in canned food products differed considerably among food types, but all were below the specific migration limit of 0.6 mg/kg set by the European Commission Directive for BPA in food or food simulants. Canned tuna products had the highest BPA concns. in general, with mean and max. values of 137 and 534 ng/g, resp. BPA concns. in the condensed soup products were considerably higher than those in the ready-to-serve soup products, with mean and max. values of 105 and 189 ng/g, resp., for the condensed soups and 15 and 34 ng/g, resp., for the ready-to-serve soups. BPA concns. in canned vegetable products were relatively low; about 60% of the products had BPA concns. of less than 10 ng/g. Canned tomato paste products had lower BPA concns. than did canned pure tomato products. The mean and max. BPA concns. were 1.1 and 2.1 ng/g, resp., for tomato paste products and 9.3 and 23 ng/g, resp., for the pure tomato products.


Levels of bisphenol A and bisphenol F in canned foods in Iranian markets
By: Rastkari, N.; Yunesian, M.; Ahmadkhaniha, R.
Bisphenol A and bisphenol F are chem. substances widely used in industry as monomers in the prodn. of epoxy resins and polycarbonates, and as antioxidants in PVC. Epoxy resins are used as inner surface coating of food and beverage cans. The contamination of cans' content by bisphenol A and bisphenol F may pose a serious threat to human because of their estrogenic activity. In this study, the concn. of bisphenol A and bisphenol F in 48 cans of different kinds of foods (corn, tomato paste, stew and tuna fish) were detd. by gas chromatog.-mass spectrometry after solvent extn. with acetonitrile and preconcn. by headspace-solid-phase microextn. (HS-SPME). Quant. anal. was carried out based on spiked calibration samples and analyses were performed for each sample in triplicate. Bisphenol F was not detected in any of the canned foods with detection limits of 0.10ug/kg There were significant differences among the mean concns. of bisphenol A in all kinds of canned foods. The mean concns. of bisphenol A increased significantly all samples after heating in (ANOVA p
Food packaging and bisphenol A and Bis(2-ethyhexyl) phthalate exposure: finding from a dietray intervention

By: Rudel, Ruthann A.; Gray, Janet M.; Engel, Connie L.; Rawsthorne, Teresa W.; Dodson, Robin E.; Ackerman, Janet M.; Rizzo, Jeanne; Nudelman, Janet L.; Brody, Julia Green

Background: Bisphenol A (BPA) and bis(2-ethylhexyl) phthalate (DEHP) are high-prodn.-vol. chems. used in plastics and resins for food packaging. They have been assocd. with endocrine disruption in animals and in some human studies. Human exposure sources have been estd., but the relative contribution of dietary exposure to total intake has not been studied empirically. Objectives: To evaluate the contribution of food packaging to exposure, we measured urinary BPA and phthalate metabolites before, during, and after a "fresh foods" dietary intervention. Methods: We selected 20 participants in five families based on self-reported use of canned and packaged foods. Participants ate their usual diet, followed by 3 days of "fresh foods" that were not canned or packaged in plastic, and then returned to their usual diet. We collected evening urine samples over 8 days in Jan. 2010 and composited them into preintervention, during intervention, and postintervention samples. We used mixed-effects models for repeated measures and Wilcoxon signed-rank tests to assess change in urinary levels across time. Results: Urine levels of BPA and DEHP metabolites decreased significantly during the fresh foods intervention [e.g., BPA geometric mean (GM), 3.7 ng/mL preintervention vs. 1.2 ng/mL during intervention; mono-(2-ethyl-5-hydroxy hexyl) phthalate GM, 57 ng/mL vs. 25 ng/mL]. The intervention reduced GM concns. of BPA by 66% and DEHP metabolites by 53-56%. Maxima were reduced by 76% for BPA and 93-96% for DEHP metabolites. Conclusions: BPA and DEHP exposures were substantially reduced when participants' diets were restricted to food with limited packaging.


Occurrence of Bisphenol A in Indoor Dust from Two Locations in the Eastern United States and Implications for Human Exposures

By: Loganathan, Sudan N.; Kannan, Kurunthachalam

Bisphenol A (BPA) is used in the prodn. of polycarbonate plastics and epoxy resins, which are used in many consumer products. Sources of human exposures to BPA include packaged and canned food products, indoor air, and dust ingestion. Information on the relative contributions of the pathways to BPA exposures is limited. In this study, we measured concns. BPA in indoor dust collected from two locations in the Eastern United States and evaluated the contribution of dust to total BPA exposures. BPA was found in 95% of the dust samples analyzed (n = 56) at concns. ranging from

Phthalates and Bisphenols Migration in Mexican Food Cans and Plastic Food Containers
By: Gonzalez-Castro, M. I.; Olea-Serrano, M. F.; Rivas-Velasco, A. M.; Medina-Rivero, E.; Ordonez-Acevedo, Leandro G.; De Leon-Rodriguez, A.
The presence of endocrine disruptors bisphenol-A, bisphenol-A-dimethacrylate, bisphenol-A-diglycidyl-ether, phthalic-acid, dibutyl-phthalate, diethyl-phthalate and dioctyl-phthalate was detd. in vegetable cans, baby bottles and microwaveable containers from the Mexican market. Gas-Chromatog.-Mass-Spectrometry was used for the identification and High-Performance-Liq.-Chromatog. with UV/Visible light and fluorescence detectors was used for the quantification. Endocrine disruptors were found in all samples. PA and DOP were the substances most commonly found, and max. concns. were 9.549 and 0.664ug/kg, resp. from a jalapeno peppers can. Bisphenol A, phthalic-acid, bisphenol-A-dimethacrylate, bisphenol-A-diglycidyl-ether, dioctyl-phtalate and dibutyl-phthalate were found in baby bottles and microwaveable containers.


Bisphenol A (BPA) in U.S. Food

By: Schecter, Arnold; Malik, Noor; Haffner, Darrah; Smith, Sarah; Harris, T. Robert; Paepke, Olaf; Birnbaum, Linda

Bisphenol A (BPA) is a chem. used for lining metal cans and in polycarbonate plastics, such as baby bottles. In rodents, BPA is assocd. with early sexual maturation, altered behavior, and effects on prostate and mammary glands. In humans, BPA is assocd. with cardiovascular disease, diabetes, and male sexual dysfunction in exposed workers. Food is a major exposure source. The authors know of no studies reporting BPA in U.S. fresh food, canned food, and food in plastic packaging in peer reviewed journals. The authors measured BPA levels in 105 fresh and canned foods, foods sold in plastic packaging, and in cat and dog foods in cans and plastic packaging. The authors detected BPA in 63 of 105 samples, including fresh turkey, canned green beans, and canned infant formula. Ninety-three of these samples were triplicates which had similar detected levels. Detected levels ranged from 0.23 to 65.0 ng/g ww and were not assocd. with type of food or packaging but did vary with pH. BPA levels were higher for foods of pH 5 compared to more acidic and alk. foods. Detected levels were comparable to those found by others. Further research is indicated to det. BPA levels in U.S. food in larger, representative sampling.


Determination of Bisphenol A Diglycidyl Ether content in foods from lacquered cans
By: Biego, G. H. M.; Yao, K. D.; Ezoua, P.; Kouadio, L. P.
Canned foods are increasingly used in food packaging. Packaging serves mainly to preserve, inform and sell foodstuffs. In order to avoid migration issues of chem. compds. from tin cans to foods, covering internal surface of the tin cans with epoxyphenolic and organosol resins is widespread. However, monomers like Bisphenol A Diglycidyl Ether (BADGE), no. among the constituents of these resins capable of migrating to foods. This chem. compd. (BADGE) is highly toxic not only for the immune, reproductive and hepatic systems but also for biomols. such as DNA, nucleic acids, proteins, and hormones. Simulation tests of migration can be used to assess the significance of BADGE migration. For this study, the migration and degrdn. tests were realized with metallic sheets and cans lacquered with epoxyphenolic or organosol resins. BADGE concns. were detd. by High Performance Liq. Chromatog. (HPLC) using a Hewlett Packard HPLC chromatograph 1050 serial equipped with an injection valve of 50 uL, a Hewlett Packard spectrophotometric UV detector serial 1050 and a Hewlett Packard integrator serial 3396. BADGE detections were made at 275 nm and compds. were sepd. on a LiChrospher 100 RP-18 (Merck, 250 ◊ 4 mm I.D., 5 um) column protected by a guard LiChrospher 100 RP-18 (Merk, 5 m) column. The mobile phase was a mixt. of methanol, water, and dichloromethane (CH3OH-H2O-CH2Cl2) according to 50%-20%-30% proportion of solvents and the flow was 1 mL.min-1. The content in BADGE ranged from 3 to 37 g.L-1. These concns. were only slightly influenced by the storage conditions (duration and temp.). The highest concns. were found in distd. water from cans analyzed just after sterilization and the lowest concns. in distd. water from cans stored at least one day before analyses. BADGE degrdn. tests in aq. environment provided an explanation to the lowest concns. of BADGE in lacquered cans. Finally, the no. of sterilization also proved to be critically important for the redn. of BADGE content in cans.

Migration of bisphenol A diglycidyl ether and its derivatives into canned meat and fish

By: Luo, Shengliang; Wu, Shaolin; Zhang, Zhaohui; Zhou, Guangyi; Gao, Yangyang; Li, Jing

A comprehensive anal. method based on high liq. chromatog.-electrospray ionization tandem mass spectrometry with pos. ionization mode was applied for measuring bisphenol A diglycidyl ether (BADGE) and its derivs. in canned meat and fish. Effects of contents in cans, storage time (6, 9 or 12 mo) and temp. (4°C, 20°C or 100°C) on migration of BADGE and its derivs. from varnish to canned contents were investigated. One-way anal. of variance (ANOVA) showed that there were significant differences among different canned contents (P
Determination of Bisphenol A and Bisphenol B Residues in Canned Peeled Tomatoes by Reversed-Phase Liquid Chromatography

By: Grumetto, Lucia; Montesano, Domenico; Seccia, Serenella; Albrizio, Stefania; Barbato, Francesco

Bisphenol A (BPA) and bisphenol B (BPB) concns. were detd. in peeled canned tomatoes of different brands bought in Italian supermarkets. Tomato samples analyzed were packaged in cans coated with either epoxyphenolic lacquer or low BADGE enamel. A solid phase extn. (SPE) was performed on C-18 Strata E cartridge followed by a step on Florisil cartridge. Detection and quantitation were performed by a reversed phase HPLC (RP-HPLC) method with both UV and fluorescence detection (FD). On the total of 42 tested tomato samples, BPA was detected in 22 samples (52.4%), while BPB was detected in 9 samples (21.4%). BPA and BPB were simultaneously present in 8 of the analyzed samples. The levels of BPA found in this study are much lower than the European Union migration limits of 3 mg/kg food and reasonably unable to produce a daily intake exceeding the limit of 0.05 mg/kg body wt., established by European Food Safety Authority.

thismomentinblackhistory: I eat a can of soup a day.

I eat a soup can a day.

bizzwire: Math time:

definitely not reading that

Canned soup is gross. Seriously, make your own and be amazed.

thelordofcheese: thismomentinblackhistory: I eat a can of soup a day.

I eat a soup can a day.

I can eat a soup a day.

bizzwire: snip

Dear BIL,

tl;dr

Love,
ur SIL

ps do you want tofurkey or field roast tomorrow?

I'm a bit surprised there's still so muched stuff canned, the boxes are becoming more common but I wonder if cans will ever lose market dominance.

/tomorrow: food boxes use baby seal livers and cesium in their manufacture!

bizzwire: [Magnets]

In the interest of full disclosure, are you employed (or derive income from) by the processed food industry?

Jarhead_h: I think the question that everyone should be asking is JUST WHY THE FRAK IS BPA IN THE CAN TO BEGIN WITH? It's not "good" for living things in ANY dose, just less harmful in smaller ones.

THIS.

The second the FDA says no more BPA, it's not like the food companies will go out of business. They got along just fine before BPA was invented.

Also, how about putting soup in glass? There's even a few companies out there selling soup in glass bottles already.

Why Yes I Am A Wizard: bizzwire: [Magnets]

In the interest of full disclosure, are you employed (or derive income from) by the processed food industry?

just a simple country analytical chemist

The primer for a generation of hypertensives who are unable to taste anything without mounds of salt.

Remember how Campbell's successfully convinced several generations of moms that it was the healthiest thing they could feed sick kids?

I don't much care for soup and rarely have it. I should be the pinnacle of health apparantly, but I'm not. It's close I guess, except for the too heavy, the gimpy foot and the sore knees if I don't wear my magic soft soled hikers.